... but pot is considerably different from alcohol. Alcohol is water soluble, canniboids are not. Irregardless of what EtOH might do when in the system, it is gone -- all gone -- in a matter of hours. Yes, there are diseases (cihhrosis for example) that are associated with excess EtOH consumption, but there's no cumulative effect.
Symposium on marijuana: Rheims, France, 22-23 July 1978
Yes, 1978.
Some tidbits:
...The inhibition of cellular proliferation and of synthesis of macromolecules by cannabinoids in eucaryote cells in culture (HeLa cells, neuroblastomas) was discussed at length at Rheims. Stein attributed this inhibiting action to an immediate action of these compounds on the plasma membrane; but, in the course of longer experiments, he suggested that the nuclear membrane might be affected particularly by the delta-8-THC of which substantial quantities are found in the nucleus. This, it seems, results in a distortion of the messages transmitted at this level by the RNA-polymerase, and a change in transcription. These observations were developed by Issidorides from Athens, who described the histochemical changes in lymphocytes and spermatozoa taken from chronic hashish smokers. These cells show a very marked deficiency in arginine-rich histones, a deficiency which is particularly evident at the level of the acrosome of the sperm cell. Such a change could be due only to a phenomenon occurring at the transcription level....
At the transcription level! That's messing with your DNA/RNA!!
Rosenkrantz reported that rats exposed to inhalation of marijuana smoke (under conditions equivalent to the daily consumption of a marijuana smoker) developed after 87 days, and up to 360 days, lesions in the lung parenchyma; these took the form of scattered small focal alveolitis, granulomatic phenomena and dense infiltrations of macrophages associated with deposits of cholesterol. The last-mentioned are signs of tissue destruction. The extent of the lesions depended on the duration of the experiments and the dose inhaled. They were still present a month after smoke inhalation had been stopped. The effects associated with marijuana were different from those produced by tobacco smoke and placebo smoke (a marijuana cigarette from which the cannabinoids had been extracted).
It's the canniboids ...
In short, it would seem that cannabinoids can act on the testicular function in two ways:
Through the gonadotropins FSH and LH, causing intermittent reductions in testosterone;
Directly on the germinative epithelium, causing changes in spermatogenesis and spermatogonia, which would explain the appearance of abnormal forms of spermatozoa.
From abnormal sperm come ...
In closing
From the public health standpoint, there are four categories of persons who might be warned forthwith of the risks involved in marjuana use. These categories are:
Adolescents, whose neuro-hormonal regulatory systems are in process of development and integration. We have seen that a single dose of marijuana can affect the secretion of the pituitary hormones referred to above;
Epilectics. The central stimulating effects of the THC may induce epileptioform seizures;
Persons with a tendency to schizophrenia and mental illness;
Women of child -bearing age who wish to have children.
From the therapeutic point of view, cannabidiol may prove to be a useful anti-epileptic nabilone, a synthetic cannabinoid, might might find some applications as an anti-emetic, as a tranquillizer and in the treatment of glaucoma. Many years will be needed to evaluate the effectiveness ot these new drugs.
I believe canniboids should be actively pharmaceuticalized. But the medical marijuna proponents have a larger agenda 
Evie 
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